Perio and Implant Blog

Blog sobre Periodoncia e Implantes Dentales

Perio and Implant Blog

Blog sobre Periodoncia e Implantes Dentales

Perio and Implant Blog

Blog sobre Periodoncia e Implantes Dentales

Perio and Implant Blog

Blog sobre Periodoncia e Implantes Dentales

Perio and Implant Blog

Blog sobre Periodoncia e Implantes Dentales

Mostrando entradas con la etiqueta socket preservation. Mostrar todas las entradas
Mostrando entradas con la etiqueta socket preservation. Mostrar todas las entradas

viernes, 30 de agosto de 2013

martes, 18 de junio de 2013

PERIODONTAL HISTORY - BECAUSE HISTORY IS FUN WITH PERIONET =)


Chinese physicians were probably first to describe signs of periodontal diseases. Diagnoses and treatments were presented in the earliest known textbook of Chinese medicine, Nei Ching, attributed to Huang Di (approximately 2700–2600 BC)
 

A N D R E W D E N T I N O , S E O K W O O L E E , J A S O N M A I L H O T & A R T H U R F. H E F T I, Principles of periodontology, Periodontology 2000, Vol. 61, 2013, 16–53

PERIODONTAL HISTORY - BECAUSE HISTORY IS FUN WITH PERIONET =)


Focal infection theory Willoughby D. Miller (1853–1907), whose work was
very much influenced by Robert Koch, is best known for his groundbreaking ideas on the etiology of caries.
He also postulated a role for bacteria in the etiology of alveolar pyorrhea , and concluded that, in the presence of predisposing factors, many bacteria found normally in the mouth can cause periodontal disease (e.g., non-specific plaque hypothesis). Miller advocated that such bacteria could also play a role in
the etiology of many other diseases in humans. He coined the expression focus of infection, but stopped short of promoting eradication of infected teeth to prevent or treat systemic illnesses. This step was made by Frank Billings (1854–1932), a highly respected American physician and academic leader. Billings and his student and colleague Edward C. Rosenow (1875–1966) promoted the theory of focal infection in the USA . Like Miller, Billings spent time in Europe, where the germ theory of disease, initiated by the groundbreaking discoveries of Koch, Lister, Pasteur and other luminaries, was heavily debated. According to Billings  theory, the germ carrier harbors pleiomorphic microorganisms that may cause disease at any time. Dissemination of such germs from a latent localized infection to a distant organ would occur through the blood and lymphatic systems. The focal infection paradigm was quickly adopted by many dentists and physicians, especially surgeons in the USA. Its clinical implementation, which was further promoted by substantial improvements in asepsis, led to uncountable unwarranted tooth extractions, tonsillectomies and other surgical procedures. In 1928, Holman publicly questioned the validity of the focal infection theory. Its importance started to decline in the 1930s as evidence accumulated indicating that surgical removal of suspected foci has no beneficial effect on the medical status of affected patients. It is tempting to speculate about apparent similarities between the focal infection theory and the so-called systemic link. However, a closer look at the basis of the two paradigms reveals fundamental discrepancies, especially with regard to bacterial pleiomorphism and tissue lesion latency.


A N D R E W D E N T I N O , S E O K W O O L E E , J A S O N M A I L H O T & A R T H U R F. H E F T I, Principles of periodontology, Periodontology 2000, Vol. 61, 2013, 16–53

domingo, 16 de junio de 2013

PERIODONTAL HISTORY - BECASUSE HISTORY IS FUN WITH PERIONET =)

Almost two centuries after Eustachi, the French surgeon-dentist Pierre Fauchard (1678–1761) published Le Chirurgien Dentiste, a two-volume book dedicated to the practice of dentistry. Fauchards comprehensive work profoundly influenced the practice of dentistry. He pointed out that any dental disease can be allocated to one of only three classes, namely: (1) diseases with external cause, (2) (hidden) diseases
that attack those tooth parts embedded in the periodontium, and (3) (symptomatic) diseases caused by the teeth. Critical of the theories accepted by most physicians of his time, Fauchard postulated a humoral etiology of periodontal disease that is
modulated by local factors, e.g. calculus. To prevent gum disease, he recommended cleaning the teeth,  massaging the gingiva with an astringent liquid, and washing the mouth with wine and water. Because of the spongy appearance of the gingiva that had similarity to gingiva he observed in patients with scurvy,
Fauchard called the phenotype "scurvy of the gums".

A N D R E W D E N T I N O , S E O K W O O L E E , J A S O N M A I L H O T & A R T H U R F. H E F T I, Principles of periodontology, Periodontology 2000, Vol. 61, 2013, 16–53

PERIODONTAL HISTORY - BECAUSE HISTORY IS FUN WITH PERIONET =)


The ancient Egyptian Ebers Papyrus was written approximately 1000 years later (approximately 1550 BC). It is one of the oldest fully preserved medical documents, and contains several passages on remedies to cure conditions such as loose teeth and swollen gums (flesh)

A N D R E W D E N T I N O , S E O K W O O L E E , J A S O N M A I L H O T & A R T H U R F. H E F T I, Principles of periodontology, Periodontology 2000, Vol. 61, 2013, 16–53

sábado, 15 de junio de 2013

PERIODONTAL HISTORY - BECAUSE HISTORY IS FUN WITH PERIONET =)



 In 1563, the Italian Renaissance anatomist and physician Bartolomeo Eustachi (1514–1574) com-
pleted  Libellus de Dentibus, the first book dedicated exclusively to the description of teeth . He was
the first to describe the periodontal ligament, as well as the deciduous and permanent dentitions. Eust-
achis profound understanding of head anatomy and his amazing eye for the detail led him to link increased tooth mobility at an advanced age with widening of the space between the root and the alveolar bone.
Similarly remarkable, he prescribed the removal of calculus and granulation tissue using scalers and cu-
rettes, respectively, to encourage re-attachment of periodontal tissues to loose teeth


A N D R E W D E N T I N O , S E O K W O O L E E , J A S O N M A I L H O T & A R T H U R F. H E F T I, Principles of periodontology, Periodontology 2000, Vol. 61, 2013, 16–53

PERIODONTAL HISTORY - BECAUSE HISTORY IS FUN WITH PERIONET =)


Early observations
Periodontal diseases, unlike caries, are not a by product of modern civilization. Manifesting themelves as ante mortem loss of alveolar bone, signs indicative of periodontal pathology were discovered in human specimens attributed to the Middle Pleisocene stage. For instance, indications of alveolar bone resorption were found in the 640- to 735,000- year-old Mauer mandible (Homo heidelbergensis) in
Germany, and in a 169- to 191,000-year-old mandible  that was unearthed at the Bau de lÕAubesier, Vaucluse, France. Such findings support the theory that
periodontal diseases have plagued humans and their phylogenetic ancestors for a very long time.

A N D R E W D E N T I N O , S E O K W O O L E E , J A S O N M A I L H O T & A R T H U R F. H E F T I, Principles of periodontology, Periodontology 2000, Vol. 61, 2013, 16–53

PERIODONTAL HISTORY - BECAUSE HISTORY IS FUN WITH PERIONET =)


In Ancient India, Sushruta (approximately 6th century BC) illustrated a large number of surgical instruments, and explained 300 surgical procedures in the treatise  Samhita. Sushruta taught a holistic approach to medical therapy. In pursuit of his philosophy, he described four periodontal conditions, offering probably the first classification of periodontal diseases


A N D R E W D E N T I N O , S E O K W O O L E E , J A S O N M A I L H O T & A R T H U R F. H E F T I, Principles of periodontology, Periodontology 2000, Vol. 61, 2013, 16–53

PERIODONTAL HISTORY - BECAUSE HISTORY IS FUN WITH PERIONET =)





Periodontal Workshop 1999
New Periodontal Classification
Periodontal History



A N D R E W D E N T I N O , S E O K W O O L E E , J A S O N M A I L H O T & A R T H U R F. H E F T I, Principles of periodontology, Periodontology 2000, Vol. 61, 2013, 16–53

miércoles, 30 de enero de 2013

PATIENT COMMUNICATION LAST PART 4





MAURO FRADEANI, REHABILITACIÓN ESTÉTICA EN PROSTODONCIA FIJA, ANÁLISIS ESTÉTICO UN ACERCAMIENTO SISTEMÁTICO AL TRATAMIENTO PROTÈSIC, VOLUMEN 1, EDITORIAL QUINTESSENCE

PATIENT COMMUNICATION PART 2



MAURO FRADEANI, REHABILITACIÓN ESTÉTICA EN PROSTODONCIA FIJA, ANÁLISIS ESTÉTICO UN ACERCAMIENTO SISTEMÁTICO AL TRATAMIENTO PROTÈSIC, VOLUMEN 1, EDITORIAL QUINTESSENCE

PATIENT COMMUNICATION PART 1








MAURO FRADEANI, REHABILITACIÓN ESTÉTICA EN PROSTODONCIA FIJA, ANÁLISIS ESTÉTICO UN ACERCAMIENTO SISTEMÁTICO AL TRATAMIENTO PROTÈSIC, VOLUMEN 1, EDITORIAL QUINTESSENCE

sábado, 8 de diciembre de 2012

DENTAL STAR WARS - EL GRAN CHEWAKA WALLPAPER


DENTAL STAR WARS
PERIODONTAL STAR WARS
STAR WARS WALLPAPER
CHEWAKA WALLPAPER
DENTAL CHEWAKA
ODONTOCHEWE
CHEWE
ODONTO CHEWAKA
ODONTOCHEWAKA
DENTAL CHEWAKA
DENTAL CHEWE

viernes, 7 de diciembre de 2012

DESARROLLO DE SITIO IMPLANTARIO CON EXTRUSIÓN ORTODÓNTICA - IMPLANT SITE DEVELOPMENT BY ORTHODONTIC EXTRUSION


Mohammed Korayema; Carlos Flores-Mirb; Usama Nassarc; Kamrin Olfertd; Implant Site Development by Orthodontic Extrusion  A Systematic Review, Angle Orthodontist, Vol 78, No 4, 2008

IMPLANT SITE DEVELOPMENT BY ORTHODONTIC EXTRUSION - DESARROLLO DE SITIO IMPLANTARIO CON EXTRUSIÓN ORTODÓNTICA

Mohammed Korayema; Carlos Flores-Mirb; Usama Nassarc; Kamrin Olfertd; Implant Site Development by Orthodontic Extrusion  A Systematic Review, Angle Orthodontist, Vol 78, No 4, 2008

DESARROLLO DE SITIO IMPLANTARIO CON EXTRUSIÓN ORTODÓNTICA - IMPLANT SITE DEVELOPMENT BY ORTHODONTIC EXTRUSION


Mohammed Korayema; Carlos Flores-Mirb; Usama Nassarc; Kamrin Olfertd; Implant Site Development by Orthodontic Extrusion  A Systematic Review, Angle Orthodontist, Vol 78, No 4, 2008

IMPLANT SITE DEVELOPMENT BY ORTHODONTIC EXTRUSION - DESARROLLO DE SITIO IMPLANTARIO CON EXTRUSIÓN ORTODÓNTICA

Mohammed Korayema; Carlos Flores-Mirb; Usama Nassarc; Kamrin Olfertd; Implant Site Development by Orthodontic Extrusion  A Systematic Review, Angle Orthodontist, Vol 78, No 4, 2008

DESARROLLO DE SITIO IMPLANTARIO CON EXTRUSIÓN ORTODÓNTICA - IMPLANT SITE DEVELOPMENT BY ORTHODONTIC EXTRUSION


Mohammed Korayema; Carlos Flores-Mirb; Usama Nassarc; Kamrin Olfertd; Implant Site Development by Orthodontic Extrusion  A Systematic Review, Angle Orthodontist, Vol 78, No 4, 2008

miércoles, 21 de noviembre de 2012

HARRY POTTER WALLPAPER - ODONTOPOTTER - GRIFFINDOR WALLPAPER - GRIFFINDENT


HARRY POTTER WALLPAPER
GRIFFINDOR WALLPAPER
ODONTOPOTTER
GRIFFINDENT
HARRY POTTER FUN
GRIFFINDOR HAT
HARRY POTTER SCARF
HARRY POTTER BROOM
HARRY POTTER TIE
HARRY POTTER FLAG
GRIFFINDOR FLAG
HARRY POTTER DIENTE
HARRY POTTER DENTISTA
HARRY POTTER DENTISTRY
HARRY POTTER TOOTH
HARRY POTTER TEETH
DIENTE DE HARRY POTTER
DIENTES DE HARRY POTTER
 , , , ,

viernes, 12 de octubre de 2012

THE PALACCI - ERICSSON CLASSIFICATION CLASS II - SOCKET PRESERVATION - CLASIFICACION DE PALACCI - ERICSSON - PRESERVACION DE ALVEOLO





The Palacci-Ericsson classification.

Vertical loss
Class I, intact or slightly reduced papillae; class II, limited loss of papillae (less than 50%); class III, severe loss of papillae; and class IV, absence of papillae (edentulous ridge).

Horizontal loss
Class A, intact or slightly reduced buccal tissues; class B, limited loss of buccal tissues; class C, severe loss of buccal tissues; and class D, extreme loss of buccal tissue, often in combination with a limited amount of attached mucosa.

Combinations of the different classes of vertical and horizontal dimensions of tissue loss

Vertical loss          Horizontal loss

Class I                        Class A
Class II                       Class B
Class III                     Class C
Class IV                     Class D

Clasificación Palacci - Ericsson

Pérdida Vertical
Clase I, papila intacta o ligeramente reducida; Clase II, pérdida de papila limitada (menos de 50%); Clase III, severa pérdida de papila; Clase IV, ausencia de papila (cresta edentula).

Pérdida Horizontal
Clase A, tejido bucal intacto o ligeramente reducido; Clase B, pérdida limitada de tejidos bucales; Clase C, pérdida sevra de tejidos bucales; Clase D, extrema pérdida de tejidos bucales, a menudo en combinación con una limitada cantidad de mucosa adherida.

Combinación de las diferentes clases de dimenciones de los tejidos verticales y horizontales

Pérdida Vertical          Pérdida Horizontal

Clase I                                 Clase A
Clase II                                Clase B
Clase III                              Clase C
Clase IV                              Clase D



PATRICK PALACCI &HESSAM NOWZARI, Soft tissue enhancement around dental implants Periodontology 2000, Vol. 47, 2008, 113–132
PERIODONTOLOGY 2000